Supplementary Figure S2 from Targeted KRASG12V Degradation In Vivo Elicits Lung Adenocarcinoma Regression with Subsequent Relapse from Dysregulated Proteolysis

Authors: Alberto Martín, Inés M. García-Pérez, Sonia San José et al.

Publication:

Published: Unknown date

Source: Crossref

Back to Search View Original Cite This Article

Abstract

<jats:p>The anti-tumor response elicited by KRASG12V degradation in immunocompromised NSG mice closely resembles the effects observed in immunocompetent C57BL/6J mice.</jats:p>

Keywords

pthe antitumor response elicited krasg12v

Supplementary Figure S2 from Targeted KRAS&lt;sup&gt;G12V&lt;/sup&gt; Degradation &lt;i&gt;In Vivo&lt;/i&gt; Elicits Lung Adenocarcinoma Regression with Subsequent Relapse from Dysregulated Proteolysis

Abstract

Keywords

Related Articles

Supplementary material to "Methodological Framework for Tsunami Risk Assessment in Port Facilities"

The Application of Interactive Digital Media Reviewed From the Cognitive Abilietes of Early Childhood Education Students Aged 4-5 Years

Data from Deep Learning Predicts Mutations and Outcomes in Gastrointestinal Stromal Tumors

Supplementary Table S4 from &lt;i&gt;Fusobacterium nucleatum&lt;/i&gt; Promotes Exosomal LncRNA &lt;i&gt;MANCR&lt;/i&gt; Secretion from Colorectal Cancer Cells to Induce PD-L1 Expression in Macrophages

Supplementary Figure S3 from Dissecting the Tumor Microenvironment to Identify Biomarkers of Outcome to Anti–PD-1 Therapy in Clear-Cell Renal Cell Carcinoma: Analyses of the HCRN GU16-260 Trial

Supplementary Figure S2 from Targeted KRAS<sup>G12V</sup> Degradation <i>In Vivo</i> Elicits Lung Adenocarcinoma Regression with Subsequent Relapse from Dysregulated Proteolysis

Supplementary Table S4 from <i>Fusobacterium nucleatum</i> Promotes Exosomal LncRNA <i>MANCR</i> Secretion from Colorectal Cancer Cells to Induce PD-L1 Expression in Macrophages